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Dhaunchak, A.-S.; Nave, K.-A.: A common mechanism of PLP/DM20 misfolding causes cysteine-mediated endoplasmic reticulum retention in oligodendrocytes and Pelizaeus-Merzbacher disease. Proceedings of the National Academy of Sciences of the United States of America 104 (45), S. 17813 - 17818 (2007)

Kassmann, C. M.; Lappe-Siefke, C.; Baes, M.; Brugger, B.; Mildner, A.; Werner, H. B.; Natt, O.; Michaelis, T.; Prinz, M.; Frahm, J. et al.; Nave, K.-A.: Axonal loss and neuroinflammation caused by peroxisome-deficient oligodendrocytes. Nature Genetics 39 (8), S. 969 - 976 (2007)

Nave, K.-A.; Sereda, M. W.; Ehrenreich, H.: Mechanisms of Disease: inherited demyelinating neuropathies - from basic to clinical research. Nature Clinical Practice Neurology 3 (8), S. 453 - 464 (2007)

Werner, H. B.; Kuhlmann, K.; Shen, S.; Uecker, M.; Schardt, A.; Dimova, K.; Orfaniotou, F.; Dhaunchak, A.; Brinkmann, B. G.; Möbius, W. et al.; Guarente, L.; Casaccia-Bonnefil, P.; Jahn, O.; Nave, K.-A.: Proteolipid Protein Is Required for Transport of Sirtuin 2 into CNS Myelin. The Journal of Neuroscience 27 (29), S. 7717 - 7730 (2007)

Krämer-Albers, E.-M.; Gehrig-Burger, K.; Thiele, C.; Trotter, J.; Nave, K.-A.: Perturbed Interactions of Mutant Proteolipid Protein/DM20 with Cholesterol and Lipid Rafts in Oligodendroglia: Implications for Dysmyelination in Spastic Paraplegia. The Journal of Neuroscience 26 (45), S. 11743 - 11752 (2006)

Meyer zu Hörste, G.; Nave, K.-A.: Animal models of inherited neuropathies. Current Opinion in Neurology 19 (5), S. 464 - 473 (2006)

Nave, K.-A.; Salzer, J. L.: Axonal regulation of myelination by neuregulin 1. Current Opinion in Neurobiology 16 (5), S. 492 - 500 (2006)

Saher, G.; Brügger, B.; Lappe-Siefke, C.; Möbius, W.; Tozawa, R.-i.; Wehr, M. C.; Wieland, F.; Ishibashi, S.; Nave, K.-A.: High cholesterol level is essential for myelin membrane growth. Nature Neuroscience 8 (4), S. 468 - 475 (2005)

Michailov, G. V.; Sereda, M. W.; Brinkmann, B. G.; Fischer, T. M.; Haug, B.; Birchmeier, C.; Role, L.; Lai, C.; Schwab, M. H.; Nave, K.-A.: Axonal Neuregulin-1 Regulates Myelin Sheath Thickness. Science 304 (5671), S. 700 - 703 (2004)

Sereda, M. W.; Meyer zu Hörste, G.; Suter, U.; Uzma, N.; Nave, K.-A.: Therapeutic administration of progesterone antagonist in a model of Charcot-Marie-Tooth disease (CMT-1A). Nature Medicine 9 (12), S. 1533 - 1537 (2003)

Lappe-Siefke, C.; Goebbels, S.; Gravel, M.; Nicksch, E.; Lee, J.; Braun, P. E.; Griffiths, I. R.; Nave, K.-A.: Disruption of Cnp1 uncouples oligodendroglial functions in axonal support and myelination. Nature Genetics 33 (3), S. 366 - 374 (2003)

Griffiths, I.; Klugmann, M.; Anderson, T.; Yool, D.; Thomson, C.; Schwab, M. H.; Schneider, A.; Zimmermann, F.; McCulloch, M.; Nadon, N. et al.; Nave, K.-A.: Axonal Swellings and Degeneration in Mice Lacking the Major Proteolipid of Myelin. Science 280 (5369), S. 1610 - 1613 (1998)

Klugmann, M.; Schwab, M. H.; Schneider, A.; Zimmermann, F.; Griffiths, I. R.; Nave, K.-A.: Assembly of CNS Myelin in the Absence of Proteolipid Protein. Neuron 18 (1), S. 59 - 70 (1997)

Sereda, M.; Griffiths, I.; Pühlhofer, A.; Stewart, H.; Rossner, M. J.; Zimmermann, F.; Magyar, J. P.; Schneider, A.; Hund, E.; Meinck, H.-M. et al.; Suter, U.; Nave, K.-A.: A Transgenic Rat Model of Charcot-Marie-Tooth Disease. Neuron 16 (5), S. 1049 - 1060 (1996)

Readhead, C.; Schneider, A.; Griffiths, I.; Nave, K.-A.: Premature arrest of myelin formation in transgenic mice with increased proteolipid protein gene dosage. Neuron 12 (3), S. 583 - 595 (1994)

Schneider, A.; Montague, P.; Griffiths, I.; Fanarraga, M.; Kennedy, P.; Brophy, P.; Nave, K.-A.: Uncoupling of hypomyelination and glial cell death by a mutation in the proteolipid protein gene. Nature 358, S. 758 - 761 (1992)

Schäffner, E.; Edgar, J. M.; Lehning, M.; Strauß, J.; Bosch-Queralt, M.; Wieghofer, P.; Berghoff, S. A.; Krueger, M.; Morawski, M.; Reinert, T. et al.; Möbius, W.; Barrantes-Freer, A.; Prinz, M.; Reich, D.S.; Flügel, A.; Stadelmann, C.; Fledrich, R.; Stassart, R. M.; Nave, K.-A.: Myelin insulation as a risk factor for axonal degeneration in autoimmune demyelinating disease. bioRxiv (2021)