Publications of Simone Eggert
All genres
Journal Article (30)
2023
Journal Article
13, 141 (2023)
APP family member dimeric complexes are formed predominantly in synaptic compartments. Cell & Bioscience
Journal Article
11, 87 (2023)
Differential effects of familial Alzheimer’s disease-causing mutations on amyloid precursor protein (APP) trafficking, proteolytic conversion, and synaptogenic activity. Acta Neuropathologica Communications 2022
Journal Article
11 (21), 3421 (2022)
The Hidden Role of Non-Canonical Amyloid β Isoforms in Alzheimer’s Disease. Cells
Journal Article
403 (1), pp. 43 - 71 (2022)
Brothers in arms: proBDNF/BDNF and sAPPα/Aβ-signaling and their common interplay with ADAM10, TrkB, p75NTR, sortilin, and sorLA in the progression of Alzheimer’s disease. Biological Chemistry
Journal Article
403 (1), pp. 3 - 26 (2022)
The role of mycotoxins in neurodegenerative diseases: current state of the art and future perspectives of research. Biological Chemistry 2021
Journal Article
3, pp. 281 - 287 (2021)
Heterologous production of a cyanobacterial bacteriocin with potent antibacterial activity. Current Research in Biotechnology 2020
Journal Article
77, pp. 5223 - 5242 (2020)
The Rab5 activator RME-6 is required for amyloid precursor protein endocytosis depending on the YTSI motif. Cellular and Molecular Life Sciences
Journal Article
26 (3), pp. 185 - 186 (2020)
Forschungskolleg “NeurodegX”. Neuroforum 2019
Journal Article
151 (5), pp. 626 - 641 (2019)
Copper and zinc ions govern the trans‐directed dimerization of APP family members in multiple ways. Journal of Neurochemistry 2018
Journal Article
55, pp. 5809 - 5829 (2018)
Trafficking in Alzheimer’s Disease: Modulation of APP Transport and Processing by the Transmembrane Proteins LRP1, SorLA, SorCS1c, Sortilin, and Calsyntenin. Molecular Neurobiology
Journal Article
75, pp. 301 - 322 (2018)
Dimerization leads to changes in APP (amyloid precursor protein) trafficking mediated by LRP1 and SorLA. Cellular and Molecular Life Sciences 2017
Journal Article
37 (21), pp. 5345 - 5365 (2017)
APLP1 Is a Synaptic Cell Adhesion Molecule, Supporting Maintenance of Dendritic Spines and Basal Synaptic Transmission. The Journal of Neuroscience
Journal Article
10, 140 (2017)
Fe65-PTB2 Dimerization Mimics Fe65-APP Interaction. Frontiers in Molecular Neuroscience
Journal Article
10, 118 (2017)
LRP1 Modulates APP Intraneuronal Transport and Processing in Its Monomeric and Dimeric State. Frontiers in Molecular Neuroscience 2014
Journal Article
8, 410 (2014)
Shedding of APP limits its synaptogenic activity and cell adhesion properties. Frontiers in Cellular Neuroscience
Journal Article
34 (33), pp. 11159 - 11172 (2014)
Amyloid Precursor Protein Dimerization and Synaptogenic Function Depend on Copper Binding to the Growth Factor-Like Domain. The Journal of Neuroscience 2013
Journal Article
248, pp. 441 - 450 (2013)
Age-dependent increases in tau phosphorylation in the brains of type 2 diabetic rats correlate with a reduced expression of p62. Experimental Neurology
Journal Article
8 (5), p. e64149 (2013)
A New Mint1 Isoform, but Not the Conventional Mint1, Interacts with the Small GTPase Rab6. PLoS One
Journal Article
33 (39), pp. 15596 - 15602 (2013)
Early BDNF Treatment Ameliorates Cell Loss in the Entorhinal Cortex of APP Transgenic Mice. The Journal of Neuroscience 2012
Journal Article
51 (1-2), pp. 43 - 52 (2012)
Amyloid precursor protein (APP) regulates synaptic structure and function. Molecular and Cellular Neuroscience