Publikationen von Klaus-Armin Nave

Beggs, H. E.; Schahin-Reed, D.; Zang, K.; Goebbels, S.; Nave, K.-A.; Gorski, J.; Jones, K. R.; Sretavan, D.; Reichardt, L. F.: FAK deficiency in cells contributing to the basal lamina results in cortical abnormalities resembling congenital muscular dystrophies. Neuron 40 (3), S. 501 - 514 (2003)

Kleppisch, T.; Wolfsgruber, W.; Feil, S.; Allmann, R.; Wotjak, C. T.; Goebbels, S.; Nave, K.-A.; Hofmann, F.; Feil, R.: Hippocampal cGMP-dependent protein kinase I supports an age- and protein synthesis-dependent component of long-term potentiation but is not essential for spatial reference and contextual memory. The Journal of Neuroscience 23 (14), S. 6005 - 6012 (2003)

Al-Saktawi, K.; McLaughlin, M.; Klugmann, M.; Schneider, A.; Barrie, J. A.; McCulloch, M. C.; Montague, P.; Kirkham, D.; Nave, K.-A.; Griffiths, I. R.: Genetic background determines phenotypic severity of the Plp rumpshaker mutation. Journal of Neuroscience Research 72 (1), S. 12 - 24 (2003)

Jurevics, H.; Hostettler, J.; Sammond, D. W.; Nave, K.-A.; Toews, A. D.; Morell, P.: Normal metabolism but different physical properties of myelin from mice deficient in proteolipid protein. Journal of Neuroscience Research 71 (6), S. 826 - 834 (2003)

Lappe-Siefke, C.; Goebbels, S.; Gravel, M.; Nicksch, E.; Lee, J.; Braun, P. E.; Griffiths, I. R.; Nave, K.-A.: Disruption of Cnp1 uncouples oligodendroglial functions in axonal support and myelination. Nature Genetics 33 (3), S. 366 - 374 (2003)

Stegmüller, J.; Werner, H.; Nave, K.-A.; Trotter, J.: The proteoglycan NG2 is complexed with α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors by the PDZ glutamate receptor interaction protein (GRIP) in glial progenitor cells - Implications for glial-neuronal signaling. The Journal of Biological Chemistry 278 (6), S. 3590 - 3598 (2003)

Erb, M.; Steck, A. J.; Nave, K.-A.; Schaeren-Wiemers, N.: Differential expression of L- and S-MAG upon cAMP stimulated differentiation in oligodendroglial cells. Journal of Neuroscience Research 71 (3), S. 326 - 337 (2003)

Rodemer, C.; Thai, T.-P.; Brugger, B.; Kaercher, T.; Werner, H.; Nave, K.-A.; Wieland, F.; Gorgas, K.; Just, W. W.: Inactivation of ether lipid biosynthesis causes male infertility, defects in eye development and optic nerve hypoplasia in mice. Human Molecular Genetics 12 (15), S. 1881 - 1895 (2003)

Rünker, A. E.; Bartsch, U.; Nave, K.-A.; Schachner, M.: The C264Y missense mutation in the extracellular domain of L1 impairs protein trafficking in vitro and in vivo. The Journal of Neuroscience 23 (1), S. 277 - 286 (2003)

Yool, D.; Montague, P.; McLaughlin, M.; McCulloch, M. C.; Edgar, J. M.; Nave, K.-A.; Davies, R. W.; Griffiths, I. R.; McCallion, A. S.: Phenotypic analysis of mice deficient in the major myelin protein MOBP, and evidence for a novel Mobp isoform. Glia 39 (3), S. 256 - 267 (2002)

Edgar, J. M.; Anderson, T. J.; Dickinson, P. J.; Barrie, J. A.; McCulloch, M. C.; Nave, K.-A.; Griffiths, I. R.: Survival of, and competition between, oligodendrocytes expressing different alleles of the Plp gene. Journal of Cell Biology 158 (4), S. 719 - 729 (2002)

Genoud, S.; Lappe-Siefke, C.; Goebbels, S.; Radtke, F.; Aguet, M.; Scherer, S. S.; Suter, U.; Nave, K.-A.; Mantei, N.: Notch1 control of oligodendrocyte differentiation in the spinal cord. Journal of Cell Biology 158 (4), S. 709 - 718 (2002)

Maier, M.; Berger, P.; Nave, K.-A.; Suter, U.: Identification of the Regulatory Region of the Peripheral Myelin Protein 22 (PMP22) Gene That Directs Temporal and Spatial Expression in Development and Regeneration of Peripheral Nerves. Molecular and Cellular Neuroscience 20 (1), S. 93 - 109 (2002)

Simons, M.; Kramer, E. M.; Macchi, P.; Rathke-Hartlieb, S.; Trotter, J.; Nave, K.-A.; Schulz, J. B.: Overexpression of the myelin proteolipid protein leads to accumulation of cholesterol and proteolipid protein in endosomes/lysosomes: implications for Pelizaeus-Merzbacher disease. The Journal of Cell Biology: JCB 157 (2), S. 327 - 336 (2002)

Garbern, J. Y.; Yool, D. A.; Moore, G. J.; Wilds, I. B.; Faulk, M. W.; Klugmann, M.; Nave, K.-A.; Sistermans, E. A.; van der Knaap, M. S.; Bird, T. D. et al.; Shy, M. E.; Kamholz, J. A.; Griffiths, I. R.: Patients lacking the major CNS myelin protein, proteolipid protein 1, develop length-dependent axonal degeneration in the absence of demyelination and inflammation. Brain 125 (3), S. 551 - 561 (2002)

Atanasoski, S.; Scherer, S. S.; Nave, K.-A.; Suter, U.: Proliferation of Schwann cells and regulation of cyclin D1 expression in an animal model of Charcot-Marie-Tooth disease type 1A. Journal of Neuroscience Research 67 (4), S. 443 - 449 (2002)

Yool, D.; Klugmann, M.; Barrie, J. A.; McCulloch, M. C.; Nave, K.-A.; Griffiths, I. R.: Observations on the structure of myelin lacking the major proteolipid protein. Neuropathology and Applied Neurobiology 28 (1), S. 75 - 78 (2002)

Werner, H.; Dimou, L.; Klugmann, M.; Pfeiffer, S.; Nave, K.-A.: Multiple Splice Isoforms of Proteolipid M6B in Neurons and Oligodendrocytes. Molecular and Cellular Neuroscience 18 (6), S. 593 - 605 (2001)

Cerghet, M.; Bessert, D. A.; Nave, K.-A.; Skoff, R. P.: Differential expression of apoptotic markers in jimpy and in Plp overexpressors: evidence for different apoptotic pathways. Journal of Neurocytology 30 (9-10), S. 841 - 855 (2001)

Nobbio, L.; Mancardi, G.; Grandis, M.; Levi, G.; Suter, U.; Nave, K.-A.; Windebank, A. J.; Abbruzzese, M.; Schenone, A.: PMP22 transgenic dorsal root ganglia cultures show myelin abnormalities similar to those of human CMT1A. Annals of Neurology 50 (1), S. 47 - 55 (2001)