Publikationen des MPI für Experimentelle Medizin bis 2021

Anderson, T. J.; Klugmann, M.; Thomson, C. E.; Schneider, A.; Readhead, C.; Nave, K.-A.; Griffiths, I. R.: Distinct Phenotypes Associated with Increasing Dosage of the PLP Gene: Implications for CMT1A Due toPMP22Gene Duplication. Annals of the New York Academy of Sciences 883 (1), S. 234 - 246 (1999)

Niemann, S.; Sereda, M. W.; Rossner, M.; Stewart, H.; Suter, U.; Meinck, H.-M.; Griffiths, I. R.; Nave, K.-A.: The “CMT Rat”: Peripheral Neuropathy and Dysmyelination Caused by Transgenic Overexpression ofPMP22. Annals of the New York Academy of Sciences 883 (1), S. 254 - 261 (1999)

Suter, U.; Nave, K.-A.: Transgenic Mouse Models of CMT1A and HNPP. Annals of the New York Academy of Sciences 883 (1), S. 247 - 253 (1999)

Coetzee, T.; Suzuki, K.; Nave, K.-A.; Popko, B.: Myelination in the Absence of Galactolipids and Proteolipid Proteins. Molecular and Cellular Neuroscience 14 (1), S. 41 - 51 (1999)

Bradl, M.; Bauer, J.; Inomata, T.; Zielasek, J.; Nave, K.-A.; Toyka, K.; Lassmann, H.; Wekerle, H.: Transgenic Lewis rats overexpressing the proteolipid protein gene: myelin degeneration and its effect on T cell-mediated experimental autoimmune encephalomyelitis. Acta Neuropathologica 97, S. 595 - 606 (1999)

Eckart, K.; Radulovic, J.; Radulovic, M.; Jahn, O.; Blank, T.; Stiedl, O.; Spiess, J.: Actions of CRF and its analogs. Current medicinal chemistry 6 (11), S. 1035 - 1053 (1999)

Dimou, L.; Klugmann, M.; Werner, H.; Jung, M.; Nave, K.-A.; Griffiths, I. R.: Dysmyelination In Mice and the Proteolipid Protein Gene Family. In: The Functional Roles of Glial Cells in Health and Disease, S. 261 - 271 (Hg. Matsas, R.; Tsacopoulos, M.). Springer US, Boston, MA (1999)