Publikationen von Klaus-Armin Nave

Simons, M.; Kramer, E. M.; Macchi, P.; Rathke-Hartlieb, S.; Trotter, J.; Nave, K.-A.; Schulz, J. B.: Overexpression of the myelin proteolipid protein leads to accumulation of cholesterol and proteolipid protein in endosomes/lysosomes: implications for Pelizaeus-Merzbacher disease. The Journal of Cell Biology: JCB 157 (2), S. 327 - 336 (2002)

Garbern, J. Y.; Yool, D. A.; Moore, G. J.; Wilds, I. B.; Faulk, M. W.; Klugmann, M.; Nave, K.-A.; Sistermans, E. A.; van der Knaap, M. S.; Bird, T. D. et al.; Shy, M. E.; Kamholz, J. A.; Griffiths, I. R.: Patients lacking the major CNS myelin protein, proteolipid protein 1, develop length-dependent axonal degeneration in the absence of demyelination and inflammation. Brain 125 (3), S. 551 - 561 (2002)

Atanasoski, S.; Scherer, S. S.; Nave, K.-A.; Suter, U.: Proliferation of Schwann cells and regulation of cyclin D1 expression in an animal model of Charcot-Marie-Tooth disease type 1A. Journal of Neuroscience Research 67 (4), S. 443 - 449 (2002)

Yool, D.; Klugmann, M.; Barrie, J. A.; McCulloch, M. C.; Nave, K.-A.; Griffiths, I. R.: Observations on the structure of myelin lacking the major proteolipid protein. Neuropathology and Applied Neurobiology 28 (1), S. 75 - 78 (2002)

Werner, H.; Dimou, L.; Klugmann, M.; Pfeiffer, S.; Nave, K.-A.: Multiple Splice Isoforms of Proteolipid M6B in Neurons and Oligodendrocytes. Molecular and Cellular Neuroscience 18 (6), S. 593 - 605 (2001)

Cerghet, M.; Bessert, D. A.; Nave, K.-A.; Skoff, R. P.: Differential expression of apoptotic markers in jimpy and in Plp overexpressors: evidence for different apoptotic pathways. Journal of Neurocytology 30 (9-10), S. 841 - 855 (2001)

Nobbio, L.; Mancardi, G.; Grandis, M.; Levi, G.; Suter, U.; Nave, K.-A.; Windebank, A. J.; Abbruzzese, M.; Schenone, A.: PMP22 transgenic dorsal root ganglia cultures show myelin abnormalities similar to those of human CMT1A. Annals of Neurology 50 (1), S. 47 - 55 (2001)

Yamada, M.; Jung, M.; Tetsushi, K.; Ivanova, A.; Nave, K.-A.; Ikenaka, K.: Mutant PLP/DM20 Cannot Be Processed to Secrete PLP-Related Oligodendrocyte Differentiation/Survival Factor. Neurochemical Research 26, S. 639 - 645 (2001)

Krämer, E.-M.; Schardt, A.; Nave, K.-A.: Membrane traffic in myelinating oligodendrocytes. Microscopy Research and Technique 52 (6), S. 656 - 671 (2001)

Schneider, S.; Bosse, F.; D'Urso, D.; Müller, H.-W.; Sereda, M. W.; Nave, K.-A.; Niehaus, A.; Kempf, T.; Schnölzer, M.; Trotter, J.: The AN2 Protein Is a Novel Marker for the Schwann Cell Lineage Expressed by Immature and Nonmyelinating Schwann Cells. The Journal of Neuroscience 21 (3), S. 920 - 933 (2001)

Yool, D.A.; Klugmann, M.; McLaughlin, M.; Vouyiouklis, D.A.; Dimou, L.; Barrie, J.A.; McCulloch, M.C.; Nave, K.-A.; Griffiths, I.R.: Myelin proteolipid proteins promote the interaction of oligodendrocytes and axons. Journal of Neuroscience Research 63 (2), S. 151 - 164 (2001)

Britsch, S.; Goerich, D. E.; Riethmacher, D.; Peirano, R. I.; Rossner, M.; Nave, K.-A.; Birchmeier, C.; Wegner, M.: The transcription factor Sox10 is a key regulator of peripheral glial development. Genes and Development 15, S. 66 - 78 (2001)

Niemann, S.; Sereda, M. W.; Suter, U.; Griffiths, I. R.; Nave, K.-A.: Uncoupling of Myelin Assembly and Schwann Cell Differentiation by Transgenic Overexpression of Peripheral Myelin Protein 22. The Journal of Neuroscience 20 (11), S. 4120 - 4128 (2000)

Schwab, M. H.; Bartholomae, A.; Heimrich, B.; Feldmeyer, D.; Druffel-Augustin, S.; Goebbels, S.; Naya, F. J.; Zhao, S.; Frotscher, M.; Tsai, M.-J. et al.; Nave, K.-A.: Neuronal Basic Helix-Loop-Helix Proteins (NEX and BETA2/Neuro D) Regulate Terminal Granule Cell Differentiation in the Hippocampus. The Journal of Neuroscience 20 (10), S. 3714 - 3724 (2000)

Klein, L.; Klugmann, M.; Nave, K.-A.; Tuohy, V. K.; Kyewski, B.: Shaping of the autoreactive T-cell repertoire by a splice variant of self protein expressed in thymic epithelial cells. Nature Medicine 6, S. 56 - 61 (2000)

Anderson, T. J.; Klugmann, M.; Thomson, C. E.; Schneider, A.; Readhead, C.; Nave, K.-A.; Griffiths, I. R.: Distinct Phenotypes Associated with Increasing Dosage of the PLP Gene: Implications for CMT1A Due toPMP22Gene Duplication. Annals of the New York Academy of Sciences 883 (1), S. 234 - 246 (1999)

Niemann, S.; Sereda, M. W.; Rossner, M.; Stewart, H.; Suter, U.; Meinck, H.-M.; Griffiths, I. R.; Nave, K.-A.: The “CMT Rat”: Peripheral Neuropathy and Dysmyelination Caused by Transgenic Overexpression ofPMP22. Annals of the New York Academy of Sciences 883 (1), S. 254 - 261 (1999)

Suter, U.; Nave, K.-A.: Transgenic Mouse Models of CMT1A and HNPP. Annals of the New York Academy of Sciences 883 (1), S. 247 - 253 (1999)

Coetzee, T.; Suzuki, K.; Nave, K.-A.; Popko, B.: Myelination in the Absence of Galactolipids and Proteolipid Proteins. Molecular and Cellular Neuroscience 14 (1), S. 41 - 51 (1999)

Bradl, M.; Bauer, J.; Inomata, T.; Zielasek, J.; Nave, K.-A.; Toyka, K.; Lassmann, H.; Wekerle, H.: Transgenic Lewis rats overexpressing the proteolipid protein gene: myelin degeneration and its effect on T cell-mediated experimental autoimmune encephalomyelitis. Acta Neuropathologica 97, S. 595 - 606 (1999)